Изследвания и информация (вътрешно ползване)
Moderator: Милко Георгиев
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Лазар Радков (Thai)
- изгонен.
- Posts: 58
- Joined: Tue Jan 11, 2005 14:44
- Location: София
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Лазар Радков (Thai)
- изгонен.
- Posts: 58
- Joined: Tue Jan 11, 2005 14:44
- Location: София
How Essential Are the Essential Fatty Acids?
The PUFA Report Part 1: A Critical Review of the Requirement for Polyunsaturated Fatty Acids
http://www.cholesterol-and-health.com/P ... eport.html
DHA била есенциална, както и АА .. ЕРА не била
както и АLA 
The PUFA Report Part 1: A Critical Review of the Requirement for Polyunsaturated Fatty Acids
http://www.cholesterol-and-health.com/P ... eport.html
DHA била есенциална, както и АА .. ЕРА не била
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Лазар Радков (Thai)
- изгонен.
- Posts: 58
- Joined: Tue Jan 11, 2005 14:44
- Location: София
високо-белтъчният прием на преебава бъбреците - даже обратното ниско-белтъчният ги преебава
http://ndt.oxfordjournals.org/cgi/content/full/20/3/657
http://ndt.oxfordjournals.org/cgi/content/full/20/3/657
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Лазар Радков (Thai)
- изгонен.
- Posts: 58
- Joined: Tue Jan 11, 2005 14:44
- Location: София
Effect of Dietary Fat and Cholesterol on Plasma Lipids and Lipoprotein Fractions in Normolipidemic Men
Conclusion: Plasma lipids were not affected either by dietary cholesterol or by any interaction of dietary fat with cholesterol.
Men Classified as Hypo- or Hyperresponders to Dietary Cholesterol Feeding Exhibit Differences in Lipoprotein Metabolism
Conclusion: These data suggest that additional dietary cholesterol does not increase the risk of developing an atherogenic lipoprotein profile in healthy men, regardless of their response classification.
Dietary Cholesterol from Eggs Increases Plasma HDL Cholesterol in Overweight Men Consuming a Carbohydrate-Restricted Diet
Conslusion: These results suggest that including eggs in a CRD results in increased HDL-C while decreasing the risk factors associated with MetS.

Conclusion: Plasma lipids were not affected either by dietary cholesterol or by any interaction of dietary fat with cholesterol.
Men Classified as Hypo- or Hyperresponders to Dietary Cholesterol Feeding Exhibit Differences in Lipoprotein Metabolism
Conclusion: These data suggest that additional dietary cholesterol does not increase the risk of developing an atherogenic lipoprotein profile in healthy men, regardless of their response classification.
Dietary Cholesterol from Eggs Increases Plasma HDL Cholesterol in Overweight Men Consuming a Carbohydrate-Restricted Diet
Conslusion: These results suggest that including eggs in a CRD results in increased HDL-C while decreasing the risk factors associated with MetS.
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Лазар Радков (Thai)
- изгонен.
- Posts: 58
- Joined: Tue Jan 11, 2005 14:44
- Location: София
Diet and Serum Cholesterol in Man
Lack of Effect of Dietary Cholesterol
Мащабен мета анализ (изследване на много изследвания), всички до едно, сочещи липсата на каквато и да била асоциация между диетичния и серумния холестерол
Lack of Effect of Dietary Cholesterol
Мащабен мета анализ (изследване на много изследвания), всички до едно, сочещи липсата на каквато и да била асоциация между диетичния и серумния холестерол
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Лазар Радков (Thai)
- изгонен.
- Posts: 58
- Joined: Tue Jan 11, 2005 14:44
- Location: София
Effects of Dietary Fat on Muscle Substrates, Metabolism, and Performance in Athletes
Conclusions: Muscle glycogen stores were maintained after a 5-wk high-fat diet period whereas IMCL content was more than doubled. Endurance performance capacity was maintained at moderate to high-exercise intensities with a significantly larger contribution of lipids to total energy turnover
Conclusions: Muscle glycogen stores were maintained after a 5-wk high-fat diet period whereas IMCL content was more than doubled. Endurance performance capacity was maintained at moderate to high-exercise intensities with a significantly larger contribution of lipids to total energy turnover
- Милко Георгиев
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Glucosamine-induced insulin resistance in L6 muscle cells.
Bailey CJ, Turner SL.
School of Life and Health Sciences, Aston University, Birmingham, UK. [email protected]
BACKGROUND: Glucosamine increases flux through the hexosamine pathway, causing insulin resistance and disturbances similar to diabetic glucose toxicity. AIM: This study examines the effect of glucosamine on glucose uptake by cultured L6 muscle cells as a model of insulin resistance. METHODS: Glucose uptake by L6 myotubes was measured using the non-metabolized glucose analogue 2-deoxy-d-glucose after incubation with glucosamine for 4 and 24 h, with and without insulin and several other agents (metformin, peroxovanadium and d-pinitol) that improve glucose uptake in diabetic states. RESULTS: After 4 h, high concentrations of glucosamine (5 x 10(-3) and 10(-2) M) reduced basal and insulin-stimulated glucose uptake by up to 50%. After 24 h, the effect of insulin was completely abolished by 10(-2) M glucosamine and reduced over 50% by 5 x 10(-3) M glucosamine. Lower concentrations of glucosamine did not significantly alter glucose uptake. The effect of glucosamine could not be attributed to cytotoxicity assessed by the Trypan Blue test. Metformin, peroxovanadium and d-pinitol, each of which increased glucose uptake by L6 cells, did not prevent the decrease in glucose uptake with glucosamine. CONCLUSION: Glucosamine decreased insulin-stimulated glucose uptake by L6 muscle cells, providing a potential model of insulin resistance with similarities to glucose toxicity. Insulin resistance induced by glucosamine was not reversed by three agents (metformin, peroxovanadium and d-pinitol) known to enhance or partially mimic the effects of insulin.
http://www.ncbi.nlm.nih.gov/pubmed/15171754
Bailey CJ, Turner SL.
School of Life and Health Sciences, Aston University, Birmingham, UK. [email protected]
BACKGROUND: Glucosamine increases flux through the hexosamine pathway, causing insulin resistance and disturbances similar to diabetic glucose toxicity. AIM: This study examines the effect of glucosamine on glucose uptake by cultured L6 muscle cells as a model of insulin resistance. METHODS: Glucose uptake by L6 myotubes was measured using the non-metabolized glucose analogue 2-deoxy-d-glucose after incubation with glucosamine for 4 and 24 h, with and without insulin and several other agents (metformin, peroxovanadium and d-pinitol) that improve glucose uptake in diabetic states. RESULTS: After 4 h, high concentrations of glucosamine (5 x 10(-3) and 10(-2) M) reduced basal and insulin-stimulated glucose uptake by up to 50%. After 24 h, the effect of insulin was completely abolished by 10(-2) M glucosamine and reduced over 50% by 5 x 10(-3) M glucosamine. Lower concentrations of glucosamine did not significantly alter glucose uptake. The effect of glucosamine could not be attributed to cytotoxicity assessed by the Trypan Blue test. Metformin, peroxovanadium and d-pinitol, each of which increased glucose uptake by L6 cells, did not prevent the decrease in glucose uptake with glucosamine. CONCLUSION: Glucosamine decreased insulin-stimulated glucose uptake by L6 muscle cells, providing a potential model of insulin resistance with similarities to glucose toxicity. Insulin resistance induced by glucosamine was not reversed by three agents (metformin, peroxovanadium and d-pinitol) known to enhance or partially mimic the effects of insulin.
http://www.ncbi.nlm.nih.gov/pubmed/15171754
Кондиционен треньор
- Милко Георгиев
- Академик

- Posts: 1083
- Joined: Sun Sep 05, 2004 21:07
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